Solmaz Etemad

Solmaz obtained her Ph.D. degree in Molecular Cell Biology from the Medical University of Innsbruck. Her graduate research was focused on differential neuronal and nuclear targeting properties of voltage gated calcium channel auxiliary β4 splice variants and their involvement in gene regulation, in the course of research she discovered a novel β4 splice variant termed β4e. She is skilled in establishing primary cultures, especially from brain and retina. In late 2015, she joined IBA and her research is namely focused on studying the developmental regulation of FAHD1 protein in various murine tissues especially in the central nervous with focus on synaptic plasticity and mitochondrial dysfunction.

E-Mail: Solmaz.Etemad@uibk.ac.at

 

Publications

1. Depletion of oxaloacetate decarboxylase FAHD1 inhibits mitochondrial electron transport and induces cellular senescence in human endothelial cell. Petit M, Koziel R, Etemad S, Pircher H, Jansen-Dürr Exp Gerontol. 2017 Mar 8; 92:7-12. doi: 10.1016/j. exger.2017.03.004.

2. Differential neuronal targeting of a new and two known calcium channel β4 subunit splice variants correlates with their regulation of gene expression. Solmaz Etemad, Obermair GJ, Bindreither D, Benedetti A, Stanika R, Di Biase V, Burtscher V, Koschak A, Kofler R, Geley S, Wille A, Lusser A, Flockerzi V, Flucher BE. Journal of neuroscience (JNeurosci). 2014 Jan 22;34(4):1446-61. doi: 10.1523/JNEUROSCI.3935- 13.2014. (Cover photo)
http://www.jneurosci.org/content/34/4.cover-expansion
https://www.i-med.ac.at/mypoint/news/679829.html

3. The juvenile myoclonic epilepsy mutant of the calcium channel β4 subunit displays normal nuclear targeting in nerve and muscle cells. Solmaz Etemad, Marta Campiglio, Gerald J. Obermair, and Bernhard E. Flucher. Channels (Austin). 2014 May 29; 8(4).

4. Investigation of tRNA and ATPase 6/8 gene mutations in Iranian ataxia telangiectasia patients. Massoud Houshmand, Sadaf Kasraie, Solmaz Etemad Ahari, Mostafa Moin, Mohammadali Bahar and Akram Zamani. Arch Med Sci. 2011 Jun; 7(3):523-7

5. Investigation tRNA (Leu/Lys) and ATPase 6 genes mutations in Huntingtion's disease. Sadaf Kasraie, Massoud Houshmand, Mohammad Mehdi Banoei, Solmaz Etemad Ahari, Mehdi Shafa Shariat Panahi, Parvin Shariati, Mohammad Bahar and Mostafa Moin. Cell Mol Neurobiol. 2008 Nov; 28(7):933-8.

6. Investigation on mitochondrial tRNA (Leu/Lys), NDI and ATPase 6/8 in Iranian multiple sclerosis patients. Solmaz Etemad Ahari, Massoud Houshmand, Mehdi Shafa Shariat Panahi, Sadaf kasraie, Mostafa Moin and Mohammad Ali Bahar. Cell Mol Neurobiol. 2007 Sep; 27(6):695-700.

7. Point mutations on Mitochondrial DNA in Iranian patients with Friedreich's Ataxia. Solmaz Etemad Ahari, Massoud Houshmand, Sadaf Kasraie, Mostafa Moin and Mehdi Shafa Shariat Panahi. Iran J Child Neurology 2007; 2(10): 41-45.

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